1.800.KIDS DOC | 1.800.543.7362

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H. William Schnaper, MD

H. William Schnaper, MD

Vice Chairman, Department of Pediatrics; Irene Heinz Given and John LaPorte Given Research Professor in Pediatrics

Professor of Pediatrics, Northwestern University Feinberg School of Medicine

Appointments & Referrals

1.800.543.7362 (1.800.KIDS DOC)

About Me

Clinical Interests: Nephrotic syndrome

Specialties, Programs & Conditions

Specialties:

Kidney Diseases >

Education & Training

Education: University of Maryland, 1975

Postgraduate Training: St. Louis Children's Hospital/Washington University School of Medicine, 1982; Mount Sinai Hospital, 1977-1978

Board Certifications: Pediatric Nephrology, General Pediatrics

Research

Research Interests: Fibrosis; chronic kidney disease; transforming growth factor

Publications

Most recent publications

Enhancing clinical trial development for pediatric kidney diseases Pediatr Res 82 (5): 727-732
Keeping the customers stratified: moving toward genetics-based treatment options in childhood NS Kidney Int 91 (4): 781-783
Hypoxia-inducible factor-1alpha promotes glomerulosclerosis and regulates COL1A2 expression through interactions with Smad3 Kidney Int 90 (4): 797-808
Serine-204 in the linker region of Smad3 mediates the collagen-I response to TGF-beta in a cell phenotype-specific manner Exp Cell Res 319 (19): 2928-37
Hypoxia-inducible factor-2alpha and TGF-beta signaling interact to promote normoxic glomerular fibrogenesis Am J Physiol Renal Physiol 305 (9): F1323-31
TGFbeta receptor I transactivation mediates stretch-induced Pak1 activation and CTGF upregulation in mesangial cells J Cell Sci 126 (Pt 16): 3697-712
Remnant nephron physiology and the progression of chronic kidney disease Pediatr Nephrol 29 (2): 193-202
Phosphatidylinositol 3-Kinase and Rab5 GTPase Inversely Regulate the Smad Anchor for Receptor Activation (SARA) Protein Independently of Transforming Growth Factor-beta1 J Biol Chem 287 (43): 35815-24
Divergent roles of Smad3 and PI3-kinase in murine adriamycin nephropathy indicate distinct mechanisms of proteinuria and fibrogenesis Kidney Int 82 (5): 525-36
Tubulointerstitial injury and the progression of chronic kidney disease Pediatr Nephrol 27 (6): 901-9

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